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The first antiviral drugs were developed in the 1960s. They were initially meant to deal with the Herpes viruses. These early drugs were found by using trial and error discovery techniques. These techniques were not very effective and it wasn’t until the 1980s with the advent of genetic sequences of viruses before current antiviral drugs were developed.
Antiviral drugs work by being targeted on a specific attribute of a virus. The most typical target attribute is one of the proteins associated with the virus. Generally this is the protein coating shell that protects the virus, like our skin help protects us from the external environment.
Another target attribute would be an enzyme secreted by the virus and not the host (mammal). Targeting this type of attribute would allow development of a drug that would be considered a broad spectrum antiviral. This would be a drug that would be effective against a number of different strains of the same, or related, virus.
Targeting a viral protein is the easier and most effective technique of the two methods set out in the last two paragraphs. However, this method has its draw backs. The main draw back is if the virus mutates then the drug will no longer be able to recognize the virus and will lose its effectiveness.
Even today the best antiviral drugs really don’t kill the virus. If they are taken soon enough they will only mitigate the symptoms. In order to do this they must be started with 24 hours or less (preferably less) of the onset of symptoms. Even then they require one to take the medication for a week or longer. These are not quick cure medications even for the virus they were made for.
A lot of virus mutate frequently. When they do their protein coating shell changes somewhat. This can be caused by environmental conditions or conditions within a host. Some viruses don’t mutate very much at all such as smallpox. For a virus that doesn’t mutate, once an antiviral drug is designed for it the drug is usually an effective agent against that virus for a long time.
Most Influenza virus mutate frequently. Each year a new flu vaccine has to be made because of this. The vaccine is made based on six, or so, of the flu virus that were active the previous year. It takes about six months to make a vaccine so it’s a guessing game. What you end up with is a vaccine that should prevent or at least reduce the symptoms for any of the six, or so, flu virus the vaccine was made for that was active the previous year. The new vaccine does not prevent you from getting the flu from a different strain, or a mutation, of another virus. As there are dozens and dozens of different flu virus a flu shot is not adequate, unfortunately it’s the best thing we have right now.
All of that is a thing of the past now, with our new line of broad spectrum antiviral agents. The big thing that sets our antivirals apart from the rest of the pack is our antivirals don’t target the virus’ proteins or its enzymes. This means that when a virus mutates into another strain our antiviral agents will still be as effective at fighting it as they were initially, while all of the antiviral drugs on the market made for that virus will lose their effectiveness against the new strain.
While it isn’t fully understood what the actual mechanism of action is with our antiviral agents their action has been deduced from past research data. The mechanism of action is that our antiviral agents are keyed for the virus’ nucleic acid chain.
Our antiviral agents are taken orally on an empty stomach. With a few minutes they are flowing in the blood stream and within 30 minutes have coated most of the cells in the host’s body. In order for a virus to damage a cell it must get close to it (most, but not all, virus must actually enter a cell to replicate). When the virus gets in close proximity to a cell that has coated itself with our antiviral agents the virus becomes contaminated with our antiviral agent. If the agent is keyed for the virus’ nucleic acid then the agent disrupts the nucleic acid chain. This disruption disables the virus from replicating and thus renders it inactive or dead for all practical purposes.
What makes our agents so effective is they carry special sugars that are present on all of the cell membranes in a healthy person. Because of this they are non-allergic and have no side effects or adverse long term effects. They can be safely taken by anyone. If a person was allergic to one of these sugars they would never have lived into adulthood. These sugars have also been taken by diabetics with no increase in their glucose levels.
The excess sugars, from a dose, are excreted from the body within 12 hours the cells retain the sugar coating for up to a week.
Nucleic acid carries the genetic code for the virus like our DNA carries our genetic makeup code. Nucleic acid is universal in all living things. The most common form of viral nucleic acid is either RNA or DNA (but a much simpler form of DNA than humans have).
We have developed two broad spectrum antiviral agents. One is keyed for Group 1 virus and the other is keyed for Group 5 virus. Group 1 virus is a Double Stranded DNA virus. Here, Group 1 refers to the Baltimore classification for virus. Group 5 virus is a Negative-Sense Single Stranded RNA virus.
Examples of Group 1 virus are: Herpes Simples Types 1 and 2, Shingles, Cytomegalovirus, Roseolovirus, African swine flu, Simian virus, Cowpox and Smallpox. Examples of Group 5 virus are: Influenza types A, B and C, the Common Cold, Swine flu (H1N1), Avian flu (Bird flu - H5N1), Hepatitis C and E, Rubella, Dengue Virus, Yellow fever, Measles, Mumps, RSV, Rabies, Ebola, Marbug, Corona, and Borna disease virus.
What makes our antiviral different? In a few words – They work!. They work extremely fast on the viruses they are keyed for. They are still very effective even if taken three days into a viral infection they are keyed for.
We are currently working with government labs to test the effectiveness of our antivirals across the entire class of both Group 1 and Group 5 viruses. So far our agents have been found to kill various virus that until now has no known drug cure. These include, Dengue Fever, Yellow Fever, Influenza types A and B, Herpes Simples types 1 and 2, Shingles and the Common Cold.
Neither the statements made on this website nor any products offered on this website have been evaluated by the Food and Drug Administration. The products and information mentioned on this site are not intended to diagnose, treat, cure, or prevent any disease. Information and statements made are for educational purposes and are not intended to replace the advice of your doctor. GlycoMeds does not dispense medical advice, prescribe, or diagnose illness. The views and nutritional advice expressed by GlycoMeds are not intended to be a substitute for conventional medical service. If you have a medical condition, see your physician of choice.
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questions & answers || articles & press || about us || policies || site map || contact us
Copyright© 2011, GlycoMeds
Web services provided by eCreations